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Pediatrics. G.examination


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Pediatrics. G.examination


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Cecylia Ambroży


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What does it mean gross motor development, fine motor development-give the examples
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gross motor - fine motor

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What are the parts of physical examination?
General state physical development psychomotor development skin lympnodes muscles bones and joints head neck chest abdomen urogenital organs neurological examination
What measurements have to be taken to asses general state?
Vital signs -HR -BP -breathing rate -temp
Demonstrate, how we examine general state. Comment it , assuming that a patient is healthy
General impression CNS condition (level of consciousness, alert and oriented) Respiratory condition (respiratory rate, NO dyspnea) Cardiovascular condition (HR, BP, NO tachycardia or bradycardia, NO cyanosis, edemas) Vital signs (HR,BP,breathing rate, temp) General state good VIDEO!
How do we divide the physical examination?
General (general state, physical development, psychomotor development, skin, lymphnodes, muscles, bones and joints) Detailed (head, neck, chest, abdomen, urogenital organs, neurological examination)
Imagine you have found lymph node on the neck of you patient. Describe it
Size Consistence: soft, tough, cohesive, splashing Tenderness, pain Fixed or movable Skin over lymph nodes- normal, reddish, hot, ulcerous Location
Describe examination of normal skin ( no abnormalities)
Turgor Color (pink-normal, cyanosis, jaundice, paleness) Dry? Skin lesions Primary morphology (macule, papule, plaque, vesicle, Petechiae, etrc.) Secondary morphology (crust, scar, , erosion, et,) Temperature Hair, nails
What are the parameters of physical development assessed during examination?
Height/length weight head circumference chest circumference body proportions
What are the elements of psychomotor development? Explain
Gross motor Fine motor Speech Social skills
What is gross motor- what are the milestones ?
Posture and locomotion lifting head in prone position (3mo) • turns from the prone to supine position (6mo) and inversely (7mo) • sitting while seated (6-7mo) • sitting up (8mo) • standing up (10mo) • walking with suport (10-11mo), on one’s own (12mo)
What is fine motor- what are the milestones
Visual-motor coordination Newborn: uncoordinated movements, infantile reflexes • 3 mo: fixing sight, following an object, own hands • 5 mo: grasping (ape-like grasp), rattling, manipulating with objects • 7 mo: scissor-like grasp, handling few objects, transferring from one hand to the other, anticipating where to fall when thrown 9 mo: pincer-like grasp, dynamic developement of manual skills 10-11 mo: can ‚draw’, build with blocks, tries to eat on one’s own
Describe physical development of 6 mths , 6 years child, use the charts
Height/length weight head circumference (equal with the chest at 6 month) chest circumference body proportions
Describe psychomotor development od 6 mths infant, 10 mths infant
6 month -gross motor (turns from prone to supine position, sitting while seated) -fine motor (ape like grasp, manipulating objects) -speech (cooing) -social skills (distinguishes between familiar persons and strengers, recognizes mother ) 10 month -gross motor (standing up, starts walking with support) -fine motor (can draw, built blocks and eat on its oun) -speech (echolalia, speech understanding) -social skills (imitates other at play, gives toys)
What are the methods of physical examination?
Inspection palpation percussion auscultation
What skin lesions can be found while examination ?
Skin lesions -Primary morphology (macule, papule, plaque, vesicle, Petechiae, etrc.) -Secondary morphology (crust, scar, , erosion, et) Abnormalities: -pale, - grey, -cyanotic, -reddened, -yellow: jaudice ( orange shade in unconjugated hiperbilirubinemia, olive- in cholestatic jaudice -caroten pigmentation( absence of yellow colour on sclera ), -brown in adrenal dysfunction -mottled skin
What does it mean gross motor development, fine motor development-give the examples
Gross motor - posture and locomotion (walking) fine motor -visual motor coordination (eating)
6 month infant girl weighs 7 kg, her length is 65 cm, head circumference is 40 cm chest circumference is 40 cm- assess physical development of this child
The chest to head circumference ratio is appropiate to age her weight is appropiate to age her length is appropiate to age
What is dyspnoea, what kinds od dyspnoea can be distinguished ?
Dificulty in breathing Inspiratory - mild Expiratory - moderate Mixed - severe
What is proper heart rate/respiratory for neonate, infant, preschool child, school child?
Neonate 100-180 infant 110-160 preschool 90-140 school child 5-12yr 80-120 school child >12yr 60-100 60 neonate 30-40 preschool 15-20 above 12
General condition parts
 Consciousness  Cardiovascular system  Respiratory system  General appearance
Quantitative disturbances of consciousness
Awake verbal pain unresponsive
Quantitative disturbances of consciousness
- Infection (encephalitis, meningitis) - Head damagePoisoning (drugs, psychoactive substances, etc.) - Bleeding into the CNS (premature babies !!) - Expansive process (tumor)
Differentiation, diagnostics for quantitative disturbances of consoiusness
1.history, accompanying symptoms (vomiting, nausea, pain, neurological symptoms (nerve palsy, etc.) 2. Additional testing: - examination of the cerebrospinal fluid (CSF) - imaging tests (ultrasound, CT) - toxicological and biochemical tests (blood, urine)
Dyspnoea is judged by?
Increased work of breathing is judged by: • nasal flaring • expiratory grunting – to increase positive endexpiratory pressure • use of accessory muscles, especially sternomastoids • retraction (recession) of the chest wall, from use of suprasternal, intercostal and subcostal muscles • difficulty speaking (or feeding).
Type of dyspnea
Inspiratory, expiratory, mixed
Dyspnoea causes...
Central (damage to the CNS (toxic, bleeding, etc.), cardiogenic, hematological, etc.
What can be the cause of dyspnoea?
Infection (upper, lower respiratory tract) • Foreign body in the respiratory tract • Chemical burn • Vascular ring • Tracheo-esophageal fistula
Differentiation diagnostics of dyspnoea
1. Interview, accompanying symptoms (fever, anorexia, cough, cyanosis, vomiting) 2. Physical examination !! 3. Additional research - imaging (lung ultrasound, chest X-ray, contrast tests, -laryngo- endoscopy, esophago-) - laboratory (parameters of inflammation, antigens, anti-bodies) - functional (functional)
Causes of cough
- Infection (upper, lower respiratory tract) - Foreign body in the respiratory tract - Vascular ring - Tracheo-esophageal fistula - Allergy - Bronchial asthma
Differentiation diagnostics of cough
1.history, accompanying symptoms (fever, anorexia, cyanosis, vomiting, rhinitis, skin changes) 2. Physical examination !! 3. Additional research - pictorial - laboratory - functional - allergic
What is syncope?
Temporary loss of consciousness due to a decrease in brain perfusion (interruption of cerebral flow for 6-8 seconds or a reduction of oxygen delivered to the brain by 20%). Syncope is rapid onset, usually resolving spontaneously and quickly (<20 s)
Causes of syncope
- Situational - Neurocardiogenic - Heart arythmia - Shock, - Hypovolemia - Anemia - Heart defect (e.g. aortic coarctation) - Stress - Migraine - Poisoning
What is the difference between true and pseudo cyanosis?
True - increased concentration of deoxygenated hemoglobin in the blood capillary (> 5 g / dL) or presence of pathological hemoglobin (most often methemoglobin> 0.5 g / dl) pseudo - abnormal pigment in the skin (drugs - chlorpromazine, amiodarone, minocycline; heavy metals - silver, gold
What is the difference between peripheral and central cyanosis?
Peripheral cyanosis (blueness of the hands and feet) may occur when a child is cold or unwell from any cause or with polycythaemia • Central cyanosis, seen on the tongue as a slate blue colour, is associated with a fall in arterial blood oxygen tension. It can only be recognised clinically if the concentration of reduced haemoglobin in the blood exceeds 5 g/dl, so it is less pronounced if the child is anaemic
Fever differentiation and diagnostics
Differentiation • Infectious diseases (urinary, respiratory, nervous, digestive) • Autoinflammatory diseases ((fever of unknown origin (FUO), recurrent fever syndrome, juvenile idiopathic cellulitis, Kawasaki disease • Diseases of the CNS (encephalopathy) Diagnostics • Interview!! • Physical examination !! • Additional tests - depending on the most probable cause
Pain rating
Visual scale Verbal (descriptive), Numeric (0-10)
Pain, diagnostic and differentiation
Diagnostics and differentiation - location, radiation, character (piercing, etc.) - accompanying symptoms - Chronic diseases any injuries, burn acute pancreatitis biliary colic, appendicitis renal colic neuralgia (including the most common trigeminal neuralgia) migraine traumatic Interview Examination of the subject (elements of the neurological examination !!)
Primary vs secondary headache vs cranial neuralgias
Primary headaches: four main groups, comprising migraine, tensiontype headache, cluster headache (and other trigeminal autonomic cephalalgias); and other primary headaches (such as cough or exertional headache). • Secondary headaches: symptomatic of some underlying pathology, e.g. from raised intracranial pressure and space-occupying lesions • Trigeminal and other cranial neuralgias, and other headaches including root pain from herpes zoster.
What is a seizure?
A seizure is a clinical event in which there is a sudden disturbance of neurological function caused by an abnormal or excessive neuronal discharge. Seizures may be epileptic or nonepileptic.
Epileptic vs non epilepleptic seizures
Epileptic -idiopathic in 80% of cases -secondary (cerebral dysgenesis, cerebral vascular occlusion, cerebral damage) -cerebral tumor neurodegenerative dissorder -neurocutaneous syndromes non-epileptic - febrile seizures - metabolic (hypoglyceamia, hypomagnesemia, hypo/hypernatremia) - head trauma - meningitis - encephalitis - poison or toxin
Febrile seizures
- 3% of children (genetic predisposition) - between 6m and 6y - brief, tonic-clonic -rapid rise in fever!
What in medical history do we need to consider when it comes to respiratory system and cardiovascular?
Respiratory system: Cough Dyspnoe Pain in chest Apnoe Hematoptysis Cyanosis Cardiovascular system: Dyspnoe Oedemas Cyanosis
What do we need to consider during physical examination when it comes to respiratory and cardiovascular system?
Respiratory system: Tachypnoe/bradypnoe Dyspnoe Cyanosis Patologic sounds upon auscultation Abnormalities on auscultation Cardiovascular system: Tachycardia /bradycardiaMurmurs Arrhytmias Abnormal apex beat
What is cough?
Cough- sudden expulsion of air -protective reflexphysiologically removing particles from airway , incidental cough- normal phenomenon • Recurent coughing- sign of disease • Most frequently manifestation of upper or lower respiratory airways
What 3 types of cough do we have?
Dry cough- usually upper respiratory tract infections • Productive cough-usually lower respiratory tract infection( bronchitis, pneumonia) • Acute caugh( <3 weeks, usually infections), persistent( 3- 8 weeks), chronic ( >8weeks)
Causes of acute cough
• Infections • Asthma • Choking • Forein body in airways • Pneumothorax
Causes of chronic cough
• Cystic fibrosis • Gastro-esophageal reflux • Tuberculosis • Bronchiectases( CF, immune deficiencies) • Impression on airways( tumor-lymphoma, vascular ring) • Foreign body in Airways • Reccurent aspiration due to oesophago-tracheal fistula• Chronic interstitial lung diseases • Chronic sinusitis
Signs of dyspnoea:
-use of accessory respiratory muscles, -nasal flaring, -grunting, -subcostal retraction - excessive movements of diaphragm Abnormal respiratory rate( bradypnoe, tachypnoe) may accompany dyspnoea
Most frequent causes of dyspnoe- respiratory system diseases
Respiratory dyspnoea • Inspiratory: upper airway( throat,larynx, trachea) • Expiratory : lower airway- bronchi • Mixed dyspnoea: alveoli
Types of dyspnoea
Respiratory dyspnea – due to respiratory tract pathology, • Cardiac dyspnea – cardiac failure • Metabolic dyspnea – usually due to acidosis • Anaemic dyspnea – due to dysfunction or low number of red blood cells • Neurognic dyspnea – secondary to neurologic system impairmen • Inspiratory dyspnea – laryngitis, larynospasm, laryngeal oedema ( anaphylaxis), foreign body in larynx • Expiratory dyspnea- asthma, wheezy bronchitis, bronchiolitis • Mixed dyspnea- pneumonia, pleuritis, pneumothorax
Pain in chest, what is it and what causes it?
Contrary to adults, children most often suffer from non-cardiac causes of chest pain (e.g. a heart attack practically does not occur in children). Causes • Pleuritis • Emphysema • Pulmonary embolism • Myocarditis • Pericarditis • Acute pancreatitis • Psychogenic causes (after exclusion of organic causes !!!)
Apnoe, what is it and what causes it?
Blockage of airflow in respiratory tract Causes • Hypertrophy of tonsils • Decreased muscle tone • Obesity • May appear also in healthy individuals Affective apnea- preceded by strong emotions
What types of apnoe do we have?
• Central- immaturity of medullar respiratory centre, decreased sensitivity to hypercapnia, mainly in premature infants • Obstructive- obstruction of upper respiratory airways, non-efectiverespiratory movements, obstructive sleep apnoea is an indication for laryngologic consutation
Hemoptysis
Rare symptom in children Mostly originate from throat/nose • Nasophrygeal infection • tonsilitis From lower respiratory tract • bronchiectases ( cystic fibrosis, immune deficiencies) • pulmonary abscess • tuberculosis • vasculitis
What is cyanosis and what types do we have?
Definition- bluish discoloration, especially of the skin and mucous membranes, due to excessive concentration of deoxygenated hemoglobin in capilary blood -> 5 g/dl Cyanosis classification • Central- decreased saturation of oxegen saturation of hemoglobin in arterial blood, predominantly visible on lips, tongue and oral cavity. • Peripheral- decreased perfusion of periphery, visible on peripheral parts of body
What dieses can cyanosis be a symptom of in respiratory and cardiovascular systems?
Diseases of respiratory tract( respiratory failure) • Foreign body of trachea, larynx • Choanal atresia • Epiglottitis • Bronchiolitis • Severe asthma attack • End stage of cystic fibrosis Diseases of cardiovascular system • Cyanotic cardiac defect (ie. Falot syndrome) • Cardiogenic shock • In all the mentioned condiotions exept cardiogenic shock central cyanosis
Tachypnoe/bradypnoe
• Abnormal( too high/too law respiratory ratein relations to age Normal respiratory rate • newborn 40-60/min • infant 30-50/min • 1-5 years 26-40/min • 6-8 years 18-30/min • older 14-20/min • Most frequently accompanies dyspnoe
Auscultation abnormalities over lungs- pathologic sounds
Rhonci and wheezes- come from bronchi as a result of their obturation due to oedema and mucus overproduction, usually present on expiration Crepitations, crackles – audible when air comes back into atelectatic vessicles (atelectasis, inflammation) best heard at the top of inspiration, typical for pneumonia Pleural friction- best heard at the end of inspiration and the beginning of expiration in pleuritis without exudate.
Auscultation abnormalities over lungs- pathologic sounds 2
Patologic bronchial sound if present over lungs, not in the typical location- lobar pneumonia • Prolonged expiration- obstruction of bronchi , wheezy bronchitis, bronchiolitis • Prolonged inspiration – narrowing of larynx, laryngitis
Percussion abnormalities
• Dull pecussion sound –presence of fluid in pleura ( pleuritis) airless fragment of lung( atelectasis), diffuse pneumonia ( loban pneumonia) • Hyperresonant sound – emphysema • Tympanic sound - pneumothorax
Vocal fremitus abnormalities
Absent or decreased- pnaumothorax, presence of fluid in pleural cavity Increased- over an airless part of lung- lobar pneumonia
Tachycardia
• Heart rate above the normal range in relation to age. May be physiologically present. In many pathologies. Compensative mechanism • 1 month – 100-180 • 2-6 month – 100-140 •6-12 month – 100-120 •2-6 years – 90-110 •7-10 years – 80-100 •11-14 years – 70-90
What types of tachycardia do we have?
Sinusal • fever, stress, anxiety • hipotension • anaemia • conditions presenting with hypoxia, respiratory failure • severe infections • hyperthyroidism, • cardiac failure, medications/stimulants • dehydration, shock Extra sinusal- cardiologic diseases
Bradycardia
• Bradycardia- heart rate lower than norm in relation to age The most common causes • Apnoe and bradycardia of premature infants • Arrhytmias and conduction disturbances • Increased intracranial pressure • Medications
Arrhythmia
Respiratory arrhythmia- physiologic in children, increasedheart rate on inspiration, dissapaers , when a child stops to breathe Arhythmias- cardiologic diseases
How do we divide cardiac murmurs?
Cardiac ( systolic, diastolic, systo-diastolic ) • Organic causes - cardiac defects • Functional – cardiac enlargment, hyperkinetic circulation • Innocent murmurs Extracardiac (pericardial friction)
Characteristics of innocent murmurs
• Loudness 1-3/6 Levine’s scale • Without any heart failure. • Proper development • Mainly systolic • Position dependent
Apex beat abnormalities
Diffuse,high, abnormally located: cardiac defects, cardiac failure,cardiomyopathies
Skin colour abnormalites
- Palor • Redness • Icterus • Cyanosis
Causes of palor
Anaemia- palor best visible on lips, auricles, conjunctiva • Infections • Dehydration • Low blood pressure- syncope • Shock
Causes of redness
Fever • Overheating • Carbon monoxide intoxication • Neonates in first days of life- skin physiologically redpolicytemia • Local redness for example over inflammed lymphnode or joint
Causes of icterus
The most common causes !!!! • Physiologic jaundice in 60% !!!! of full term neonates • Breast milk jaundice Blood and liver diseases as causes of jaundice will be discussed next year
Patomechanisms of physiologic jaundice
Immaturity of the liver- decreased conjugation with glucuronic acid • Increased destruction of erythrocytes( presence of fetal hemoglobin,) • Increased enterohepatic circulation of bilirubin
Criteria of physiologic jaundice
• Unconjugated bilirubin • Not in the first day of life • Not longer than 10 days ( ful term newborns),14 days premature neonates • Level lower than 15 mg% (artificial feeding) i 17 mg% ( brest feeding) • The daily increase lower than 5 mg% , less than 0,3 mg% per hour • No pathologic symptoms present
Breast milk jaundice
Components of breast milk hinder bilirubin conjugation • early ( 2-3 day) late(7 day) • unconjugated bilirubin • therapeutic-diagnostic trial- the decrease in bilirubin level after stopping breast feeding for 24-48 h or feedingwith previusly heated breast milk • needs to be differentiated with pathologic jaundices
Oedema patomechanism
• Decreased oncotic pressure : (hypoalbuminemia) • Increase blood vessels permeability • Venous stasis • Lymph stasis
Main causes of oedemas
Cardiac failure • Renal failure • Liver failure • Nephrotic syndrome • Allergy
Primary skin lesions
Macule • papule • vesicle • blister • pustule • wheal • nodule
Secondary skin leasions
• crust • erosion • scale • lichenication • scar • excoriation
Macule
Most common category erythematous due to blood vessels dilatation upon pressing dissapear if its macular rash upon pressing do not dissapear if its hemorrhagic rash - fever and measles - allergic rash allergy to drugs - in Schoenlein Henoch disease - hemorrhagic rash in meningococcal sepsis - cafe au lait
Papule
- diper dermititis
Vesicle
Chickenpox (vesicular rash) wheal
Lichenication
Atopic dermatitis
Excoriations
Atopic dermititis
Scale
Seborrhoic dermatitis
During the examination of lymph nodes the following elements have to be assessed
Size location number single/cluster consistency- hard, soft, firm mobile/fixed Tenderness appearence of the skin over lymphnode: normal/inflammed
The following groups of lymph nodes have to be palpated
• occipital • retroauricular • nuchal • preauricular • cervical • submadibular • axilar • supraclavicular • inguinal
Lymphadenopathy, the most important causes
Lymphadenitis- painful lymph nodes • Reactive lymphadenopathy due to infection in the region of lymph node: submandibular lymphadenopathy in tonsilitis or labial heres • General infections( rubella- occipital lymphadenopathy, glandular fever- cervical lymphadenopathy) • Hematologic malignancies: -General lymphadenopathy- lymphoblastic leukemia -Single lymphnode or cluster- lymphoma • Non- infectious disease – Kawasaki disease
Nerons neck
Lynphadenopathy in glandular fever caused by Epstein Bar virus (mononucleosis)
Hodgkin lymphoma
Lymphadenopathy, cancer
Abdominal pain depends on....
- Type of stimuli • Type of nociceptors • Way of conduction • Patient’s personality • Cultural patterns • Other
Etiology of abdominal pain
Diseases of digestive tract • Diseases of other intraabdominal organs (kidney, urinary tract) • Disease of organs, systems located outside the abdomen (pleura, lung) • Systemic diseases (endocrine, metabolic) • Effect of drugs • Effect of toxic substances - organic diseases - functional disorders
Acute abdominal pain – surgical
- Acute pain • Vomiting • Abdominal distention • No gases, no stool, bloody stool • Fever • Severe general state • Peritoneal symptoms
Acute abdomen causes
Acute appendicitis • Ileus • Intussusception • Volvulus • Incarceretion of hernia • Peritonitis • Perforation • Meckel’s divericulitis • Acute pancreatitis • Trauma